MS Medical Education Istanbul

M
“As promised, my slides from the meeting I posted on 2 days ago.”





“The case study is the one I used at the ABN to warn against simply using clinical markers of disease activity to monitor a response to a DMT. We have to start using MRI and to act on it when we see that MSers are not responding. I also made the point that we can’t let MSers cycle through multiple 1st-line therapies that are only moderately effective we need to get them onto highly active DMTs as soon as possible. Treat early! Treat aggressively!”

About the author

Prof G

Professor of Neurology, Barts & The London. MS & Preventive Neurology thinker, blogger, runner, vegetable gardener, husband, father, cook and wine & food lover.

5 comments

  • Hope you made it home safely from Istanbul. Apparently the potests in Istanbul were directed against neurologists who keep prescribing ineffective treatments such as copaxone and beta-interferon.

    Just watched some interviews from the CMSC conference in Florida. One neuro said thathe and others were now questioning the approach of treating MS with the current first line therapies. He said he aim should be to preserve as many brain cells as possible, and treating early / aggressively was probably the best way to achieve this. Looks like the word is spreading!

    • We know the protests are over something else 🙂

      Early and agressive….yes hopefully UK MSers will get the option to try this.

  • Anonymous Saturday, June 01, 2013 9:32:00 pm

    Posted: This may be of interest
    web reference….although it is the press release not the conference paper.

    As the link requires a sign up to view it has been deleted..sorry

  • It was an article on Medscape.com entitled "Vitamin D Benefit in MS: Molecular, Genetic Evidence" The article was about a poster at: 5th Cooperative Meeting of the Consortium of Multiple Sclerosis Centers (CMSC) and the Americas Committee for Treatment and Research In Multiple Sclerosis (ACTRIMS). Poster DX10. Presented May 30, 2013. It said "The analysis showed that genes associated with increased vitamin D levels — 25-hydroxyvitamin D or 25(OH)D — and with interferon β-1b are also associated with a decrease in gadolinium-enhancing lesion (Gd+ lesion) counts." I does not show a causal link.

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