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  • I think with the current therapies, ms can be manageable if you are RRMS. In the progressive disease, MS become a mitochondrial dysfunction. I think the answer for early progressive ms lies here:

    LINK removed

    • Why did you remove the link? I thought the point was to allow us to give the Web address to the next thing. It kinda defeats the purpose.

    • The link was to the United Mitochondrial Disease Foundation. This is a foundation that supports people with mitochondrial diseases. The link I posted before was long and went directly to the treatments page, which may have raised some suspicions. This foundation does not sell anything, they are just trying to help people with this disease.

      Recently, many researchers are discovering that mitochondrial changes occur in ms which may cause neuro-degeneration or progressive disease. This foundation acknowledges the link.

      If you do a search for "United Mitochondrial Disease Foundation" you will find their page and decide for yourself.

  • We spend half our time removing links to penis extensions' cosmetic BoTox; viagra; visit my site. buy my product.

    When you add links without explanation they all need to be read and checked out….The link you posted…….Asks for money. If we leave the link, is it an endorsement? Are we being duped into sending readers there so the site can make money.

    Maybe once we have time to properly read the information in the link then the post may be removed from spam

  • Spreading hope is a therapeutic strategy that I use all the time. I was taught this term by a great clinical teacher who was a pain expert. He used to tell people with chronic pain that there 10 treatments that I would like to try and that we need to try all of them one at a time until we find the one that works for you. He called the list hope and it worked more often than not. The same principle applies to MS. With the long list of current and emerging treatments and the relentless pace of human innovation it is a matter of time before we crack the problem. The downside in this argument is the time factor; some people don't have much time. Me included. I am about to turn 50 and life seems to running away from me.

  • I had a CCSVI treatment, and after 9 months I still feel fine. It was not a miracle, it was just a simple operation that made me feel OK. Now I can wear shoes and I can walk thousand times better than before. I repeat it, it was not a miracle, but for me, it was a revolution!!!
    I deeply agree with Doctor G. Spreading hope can be therapeutic.
    This is my motto: Be happy or die trying 🙂
    Robi RR MS
    (sorry for my bad English, I have tried my best)

    • You English is very good and better than our what ever other language it is besides English.

      Hopefully we will get some news of whether your experience is consistent when the trials are announced.

  • Well, this seems to be an interesting article on MS modalities and therapy since it seems to relate. Another Black Swan,doctor?

    Does Disease-Irrelevant Intrathecal Synthesis in Multiple Sclerosis Make Sense in the Light of Tertiary Lymphoid Organs?

    We therefore hypothesize that intrathecal immune suppression should negate intrathecal IgG secretion by clearing CNS TLO (1). Clinical trials using intrathecal drugs offer a real hope to cure progressive MS. However, these trials were designed to use intrathecal rituximab. A pitfall of this treatment may be the resistance of CD20+ B-cells nested in TLO as observed in rheumatoid arthritis, and the persistence of local IgG secretion as observed in serum

    Although typical of MS, the non-specific intrathecal reaction has long been a theoretical issue. We propose to consider this non-specific reaction as a common physiological property of intrathecal inflammation, involving the general properties of TLO located in the CNS. We extend this concept to (non-MS) CNS autoimmune and infectious disorders associated with intrathecal synthesis. Moreover, we propose that the cortical lesions associated with TLO observed in MS are not specific for MS but for TLO. We therefore propose the concept of “TLO-pathy” to describe cortical lesions associated with the presence of TLO. The concept of non-specific secretion deserves further study to clarify the influence of immunization schedule.

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