6th Research Day

Are you thinking of coming? 

If you have not registered please note places are going like hot cakes.

We are trying another format this year, which limits numbers,
Remember it is a research day and it will be around our interests with some other bits put in the mix.

We will be holding the 6th MS Research Day on Saturday the 21st March 2015 at the Church House Conference Centre in Westminster, London.

We are aware that people who have attended our previous research days may already know alot about MS (nerve signals, immune response, how clinical trials work etc.) therefore, we are going to run a beginners talk from 10.00am -10.30am for people who are new to MS research and would like to learn about the basics. The official welcome will then start at 10.30am and then the research talks will follow.

If you are a regular attender and would not like to hear the talk about the basics then please still come along and join us for a coffee in one of the other rooms from 10am – 10.30am.


You can register for the event here: www.msresearchday2015.eventbrite.co.uk

If you would like to attend the event in a professional capacity (i.e you are a Neurologist, MS Nurse, researcher, medical student etc.) then please contact us before you register as there are very limited spaces.

If you are are planning to attend to represent a charity or a company please also contact us and do not use the event brite page.

We look forward to seeing you in March.

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  • Thanks MD. I hope that stress and anxiety and MS are discussed. How both stress and anxiety can exacerbate MS symptoms and trigger relapses.

  • Team G, so in my experience and it seems other MSers that anxiety triggers a relapse. I would like to know what the mechanism is for this. I have been researching anti anxiety drugs and their mechanism of action. The below mentions in anxiety disorders the central nervous system is often over active.

    About Anti-Anxiety Drugs

    There are several different classes of anti-anxiety drugs, each of which has its own mechanism of action.


    Benzodiazapenes such as Klonopin, Xanax and Ativan have sedative and anticonvulsant properties. They depress the central nervous system by acting on the receptors of the neurotransmitter GABA. The benzodiazapene binds to the receptor, causing it to have a higher affinity for GABA, which acts as an inhibitory neurotransmitter. This causes depression of the central nervous system, which is often overactive in those with anxiety disorders. Benzodiazapenes have the potential for addiction and are sometimes used as recreational drugs.


    Other anti-anxiety drugs work on the neurotransmitter serotonin. Serotonin plays a role in multiple physiological processes and has effects on sleep, appetite, mood, metabolism and body temperature. The drug BuSpar is thought to work on a specific serotonin receptor, 5-HT1A, and potentiates serotonin's inhibitory central nervous system effects, decreasing anxiety and promoting normal sleeping and eating patterns. Selective serotonin reuptake inhibitors (SSRIs), some of which are indicated for anxiety, increase the levels of serotonin in the brain instead of working on the receptors.


    Barbituates are strong central nervous system depressants. Like benzodiazapenes, they increase the inhibitory effects of GABA, but they also block the receptors for glutamate, which is an excitatory neurotransmitter. This makes barbituates extremely potent sedatives, and they can be highly addictive. Barbituates are usually only prescribed for very short periods of time, and only if other drugs have failed.

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